Of note, statistical mediating factors are implant length, diameter, and surface as well as bone quantity D vs. A (Bone quantity relates to the bone volume present. Division A is the height of the bone more than 10 mm. Division B is more than 10 mm in height, but the width at the crest is 2.5–5 mm. Division C is less than 10 mm in height and width atrophied to less than 2.5 mm. Division D is severely deficient bone. Both Division C and D will require augmentation procedures) and bone quality 4 in relation to 1 (Bone quality relates to density present. Type 1 dense bone provides great cortical anchorage but limited vascularity. Type 2 bone is the best bone for osseointegration. Type 3 and 4 bone have soft bone textures with the least success of implant integration in type 4 bone) using Lekholm and Zarb classification.

However, given a population of 98 bruxers and matched non-bruxers, considering these variables, the odds ratio of implant failure has been shown to be 2.71 in relation to non-bruxers. Furthermore, Chitumalla et al. in a recent 5-year retrospective study reported a survival rate of dental implants with bruxism habit was 90% after 1 year, 87% after 2 years, 85% after 3 years, 75% after 4 years, and 72% after 5 years. Other investigators have corroborated the link between parafunction and implant failure. This underscores the link between occlusal overload and peri-implant disease and ultimately failure. It is likely that forces applied to implants during bruxism are even higher than those exerted onto natural teeth due to the decreased proprioception of implants. The periodontal ligament of natural teeth provides the central nerve system with feedback for sensory and motor control vs. the implant having feedback from only distant mechanoreceptors and therefore lowers tactile sensitivity (8-fold!).